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D-ASPARTIC ACID

Clinically Proven to Increase Testosterone by 50% In 12 Days!

I just wanna add my 2 cents before you go any further. This product works and should be cycled 12 days on 12 days off don't be tempted to go beyond 12 days without a break

Recent research has shown the D-aspartic acid serves as a specialized neurotransmitter in parts of the nervous system involved in hormone production. It has been shown to stimulate the release of (LH) Luteinizing Hormone and (GH) Growth Hormone from the pituitary gland. Furthermore, it has also been demonstrated to have a direct stimulating effect in the testes upon testosterone production.

In Males LH acts on the interstitial cells (also known as Leydig cells) of the testes stimulating them to synthesize and secrete the male sex hormone, testosterone.

Growth hormone (GH) is a protein-based peptide hormone. It stimulates growth, cell reproduction and regeneration in humans and other animals. Growth hormone is a 191-amino acid, single-chain polypeptide that is synthesized, stored, and secreted by the somatotroph cells within the lateral wings of the anterior pituitary gland

Background

D-aspartic acid is an amino acid present in neuroendocrine tissues of invertebrates and vertebrates, including rats and humans. Here we investigated the effect of this amino acid on the release of LH and testosterone in the serum of humans and rats. Furthermore, we investigated the role of D-aspartate in the synthesis of LH and testosterone in the pituitary and testes of rats, and the molecular mechanisms by which this amino acid triggers its action.

Methods

For humans: A group of 23 men were given a daily dose of D-aspartate for 12 days, whereas another group of 20 men were given a placebo. For rats: A group of 10 rats drank a solution of either 20 mM D-aspartate or a placebo for 12 days. Then LH and testosterone accumulation was determined in the serum and D-aspartate accumulation in tissues. The effects of D-aspartate on the synthesis of LH and testosterone were gauged on isolated rat pituitary and Leydig cells. Tissues were incubated with D-aspartate, and then the concentration (synthesis) of LH and cGMP in the pituitary and of testosterone and cAMP in the Leydig cells was determined.

Results

In humans and rats, sodium D-aspartate induces an enhancement of LH and testosterone release. In the rat pituitary, sodium D-aspartate increases the release and synthesis of LH through the involvement of cGMP as a second messenger, whereas in rat testis Leydig cells, it increases the synthesis and release of testosterone and cAMP is implicated as second messenger. In the pituitary and in testes D-Asp is synthesized by a D-aspartate racemase which convert L-Asp into D-Asp. The pituitary and testes possesses a high capacity to trapping circulating D-Asp from hexogen or endogen sources.

Conclusion

D-aspartic acid is a physiological amino acid occurring principally in the pituitary gland and testes and has a role in the regulation of the release and synthesis of LH and testosterone in humans and rats.

Tested D-ASPARTIC is comprised of 100% D-Aspartic Calcium Chelate. No cheap fillers are added. 

Both men and women can suffer from too much Estrogen!

Whether brought on naturally, by the abuse of steroids, or sport nutrition test boosters Estrogen can promote and accelerate the development of breast cancer in women and cause men a number of health problems.

In men, excessive estrogen can cause gynecomastia (enlargement and formation of female like breasts in men) by binding to estrogen receptors in the nipples area.

Tested Nutrition Anti-Estro

Estrogen may also interfere with testosterone production in men and may cause libido (sex drive) problems. Other side effects of excessive estrogen include: water retention (edema), impotence (in men) and acne. The highest concentration of estrogen receptors in the body is found in the breasts and nipple area. That is the reason for potential gynecomastia problems in men and breast cancer in women. Tested Nutrition Anti-Estro works by binding to the estrogen receptors and by doing so, they prevent harmful estrogen from binding to these receptor sites. 



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